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Fat old guys
  • So I went to the YMCA yesterday using a trial pass to go sauna. The sauna was fantastic but I was the only one wearing swimming attire. The rest were buck naked. There were a few 50 or so year olds that were significantly overweight, they looked diabetic. Each of them had regular sized balls with an almond riding around on top of them. One of them didn't even look like an almond, it looked like a belly button.

    Does anyone know why this happens? There were several other 50 year old men that were in good shape and their penis's looked normal.
  • darwindarwin
    Posts: 1,196
    their cocks shrunk in terror of your youth and splendor

    darwin
  • Fat cells produce estrogen. In other words, fat feminizes. Extremely
    obese men may suffer erectile dysfunction as a result.
  • gabrial
    Posts: 31
    That is so depressing. the battle of the bulge is lost one manly rack at a time.
    gabrial
  • darwindarwin
    Posts: 1,196
    [quote=Gaston LeGris]Fat cells produce estrogen. In other words, fat feminizes. Extremely
    obese men may suffer erectile dysfunction as a result.

    are you saying their penis shrinks? i would want to see specific references before being persuaded of that. a simpler explanation is that it is buried in surrounding fat.

    see http://www.obesitycures.com/obesity-and-penis-size.html

    darwin
  • Pan
    Posts: 249
    This makes sense except... Can anyone explain how this theory applies to Ron Jeremy?

    :wink:
  • B MayfieldB Mayfield
    Posts: 2,077
    Pan said:

    This makes sense except... Can anyone explain how this theory applies to Ron Jeremy?

    :wink:



    He's a Hedgehog... LMAO :lol:
  • Pan
    Posts: 249
    Oh, of course! Thanks B. Now it makes sense. :lol:
  • OptimusOptimus
    Posts: 25
    I'm not a doctor nor do I play one on TV ... but I think it's safe to say there are at least a half-dozen biochemical markers for the X-Syndrome. Low free T3 (the thyroid hormone); high rT3 (reverse T3, an inactive thyroid hormone usually seen in starvation syndromes); disregulation of leptin; high E2 (a form of estrogen); high SHBG (sex hormone binding globulin); high cortisol; and often low testosterone.

    I know, that wasn't the answer you were looking for. :-) This syndrome isn't funny ... if nothing else, it's a sign of how the American "health-care" system is badly broken, and that's REALLY not funny. :-(
  • rookrook
    Posts: 1,604
    [QUOTE=Gaston LeGris;75869]Fat cells produce estrogen. In other words, fat feminizes. Extremely
    obese men may suffer erectile dysfunction as a result.

    Truth! The process is aromatization and is most pronounced in belly (spare tire) fat.

    Oversimplification of what happens next:

    The resulting estrogen is interpreted by the pituitary as just another sex hormone and it sends less lutenizing hormone to the testes to produce Testosterone. -- Luteinizing hormone - Wikipedia, the free encyclopedia --

    Lower Testosterone levels result in fewer nocturnal erections which eventually results in fibrosis, the principal cause of the loss of penis length and erectile quality.

    The increased Estradiol (E2) hits the prostate and is thought, over a long period to cause prostate enlargement (aka bph), which several of us in this group work to tame. Larger amounts of E2 cultivate breast growth.

    There ya got it, your basic 75-cent explanation of the success of Viagra et al.

    Oh yeah, there are a couple of aromatase inhibitors that slow/stop the conversion of "T" into Estrogens. One is Arimidex which is prescribed by Endo docs to some guys with the hope that it will increase LH and in turn boost Testostone. Oncologists also prescribe it to post-menopausal breast cancer patients. It halts the conversion of androgens produced in the adrenals into Estrogen, denying the breast tumor the Estrogen it craves.

    Bottom line, ..... avoid a spare tire !!!
  • xhepera
    Posts: 48
    [QUOTE=rook;88107]. . .Oh yeah, there are a couple of aromatase inhibitors that slow/stop the conversion of "T" into Estrogens. One is Arimidex which is prescribed by Endo docs to some guys with the hope that it will increase LH and in turn boost Testostone. Oncologists also prescribe it to post-menopausal breast cancer patients. It halts the conversion of androgens produced in the adrenals into Estrogen, denying the breast tumor the Estrogen it craves.

    . . .

    Good info. I'm not sure Arimidex is useful for raising LH though. Clomid and Nolvadex will, but the jury's still out on whether the action of Armidex will do the same. Arimidex will cause an increase in T simply by inhibiting E2 production, thereby stopping excess E2 from latching on to T receptors and tricking the brain into believing that the body has sufficient T.

    E2 is also a problem for people who have too much Testosterone. The belly fat is often a giveaway but it also happens in bodybuilders who supplement T. It was, in fact, bodybuilders who were the first to use Arimidex for this purpose. I use Arimidex because I supplement T and use HCG to mitigate the effects of T supplementation on the testes. HCG (human chorionic gonadotropin) can also accent aromatization. By the way, my use of said substances is entirely under medical supervision as I have a testosterone deficit. You mentioned that endos often prescribe it. I was lucky. . .my urologist prescribed it (at my request) and my endo (I'm also diabetic) agrees with its use. I take .25 mg (quarter pill) every 2-3 days. You have to be careful with Arimidex as going too low on E2 is every bit as bad as having too much.

    Anyway, I was gonna say that I was lucky my docs went along with the Arimidex and HCG regimen. Most will not. I'm on a hypogonadism forum filled with horror stories of guys who are being doomed to health problems and life-threatening issues because uros and endos won't prescribe Arimidex or HCG.